tailieunhanh - Báo cáo khoa học: " Effects of epinephrine and vasopressin on end-tidal carbon dioxide tension and mean arterial blood pressure in out-of-hospital cardiopulmonary resuscitation: an observational study"

Tuyển tập các báo cáo nghiên cứu về y học được đăng trên tạp chí y học Critical Care giúp cho các bạn có thêm kiến thức về ngành y học đề tài: Effects of epinephrine and vasopressin on end-tidal carbon dioxide tension and mean arterial blood pressure in out-of-hospital cardiopulmonary resuscitation: an observational study. | Available online http content 11 2 R39 Research Effects of epinephrine and vasopressin on end-tidal carbon dioxide tension and mean arterial blood pressure in out-of-hospital cardiopulmonary resuscitation an observational study Stefan Mally Alina Jelatancev and Stefek Grmec Centre for Emergency Medicine Maribor Ljubljanska 5 2000 Maribor Slovenia Corresponding author Stefan Mally Received 17 Oct 2006 Revisions requested 22 Nov 2006 Revisions received 28 Feb 2007 Accepted 21 Mar 2007 Published 21 Mar 2007 Critical Care 2007 11 R39 doi cc5726 This article is online at http content 11 2 R39 2007 Mally et al. licensee BioMed Central Ltd. This is an open access article distributed under the terms of the Creative Commons Attribution License http licenses by which permits unrestricted use distribution and reproduction in any medium provided the original work is properly cited. Open Access Abstract Introduction Clinical data considering vasopressin as an equivalent option to epinephrine in cardiopulmonary resuscitation CPR are limited. The aim of this prehospital study was to assess whether the use of vasopressin during CPR contributes to higher end-tidal carbon dioxide and mean arterial blood pressure MAP levels and thus improves the survival rate and neurological outcome. Methods Two treatment groups of resuscitated patients in cardiac arrest were compared in the epinephrine group patients received 1 mg of epinephrine intravenously every three minutes only in the vasopressin epinephrine group patients received 40 units of arginine vasopressin intravenously only or followed by 1 mg of epinephrine every three minutes during CPR. Values of end-tidal carbon dioxide and MAP were recorded and data were collected according to the Utstein style. Results Five hundred and ninety-eight patients were included with no significant demographic or clinical differences between compared groups. Final end-tidal

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