tailieunhanh - Báo cáo khoa hoc:" Late presentation of superior mesenteric artery syndrome following scoliosis surgery: a case report"

Tuyển tập báo cáo các nghiên cứu khoa học quốc tế ngành y học dành cho các bạn tham khảo đề tài: Late presentation of superior mesenteric artery syndrome following scoliosis surgery: a case report | Journal of Medical Case Reports BioMed Central Case report Open Access Late presentation of superior mesenteric artery syndrome following scoliosis surgery a case report Athanasios I Tsirikos 1 2 Raymond E Anakwe1 and Alexander DL Baker1 Address 1Scottish National Spine Deformity Centre Royal Hospital for Sick Children Edinburgh UK and 2Consultant Orthopaedic and Spinal Surgeon Honorary Clinical Senior Lecturer-University of Edinburgh c o Scottish National Spine Deformity Centre Royal Hospital for Sick Children Sciennes Road Edinburgh EH9 1LF UK Email Athanasios I Tsirikos - atsirikos@ Raymond E Anakwe - Alexander DL Baker - alexbaker@ Corresponding author Published 19 January 2008 Received 18 June 2007 Journal of Medical Case Reports 2008 2 9 doi 1752-1947-2-9 Accepted 19 January 2008 This article is available from http content 2 1 9 2008 Tsirikos et al licensee BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License http licenses by which permits unrestricted use distribution and reproduction in any medium provided the original work is properly cited. Abstract Introduction Obstruction of the third part of the duodenum by the superior mesenteric artery SMA can occur following surgical correction of scoliosis. The condition most commonly occurs in significantly underweight patients with severe deformities during the first few days to a week following spinal surgery. Case presentation We present the atypical case of a patient with normal body habitus and a 50 adolescent idiopathic thoracolumbar scoliosis who underwent anterior spinal arthrodesis with instrumentation and developed SMA syndrome due to progressive weight loss several weeks postoperatively. The condition manifested with recurrent vomiting abdominal distension marked dehydration and severe electrolyte disorder. Prolonged