tailieunhanh - Báo cáo khoa hoc:" An extended association screen in multiple sclerosis using 202 microsatellite markers targeting apoptosis-related genes does not reveal new predisposing factors"

Tuyển tập báo cáo các nghiên cứu khoa học quốc tế ngành y học dành cho các bạn tham khảo đề tài: An extended association screen in multiple sclerosis using 202 microsatellite markers targeting apoptosis-related genes does not reveal new predisposing factors | Journal of Negative Results in BioMedicine BioMed Central Research Open Access An extended association screen in multiple sclerosis using 202 microsatellite markers targeting apoptosis-related genes does not reveal new predisposing factors René Godde 1 Stefanie Brune1 Peter Jagiello1 Eckhart Sindern2 Michael Haupts3 Sebastian Schimrigk4 Norbert Muller5 and JorgT Epplen1 Address Department of Human Genetics Ruhr-University Bochum Germany 2Department of Neurology Kliniken Bergmannsheil Ruhr-University Bochum Germany 3Department of Neurology Knappschaftskrankenhaus Ruhr-University Bochum Germany 4Department of Neurology St. Josef-Hospital Ruhr-University Bochum Germany and 5Department of Transfusion Medicine Universitatsklinikum Essen Essen Germany Email René Godde - Stefanie Brune - Peter Jagiello - Eckhart Sindern - Michael Haupts - Sebastian Schimrigk - Norbert Muller - Jorg T Epplen - Corresponding author Published 05 September 2005 Received 10 March 2005 . . 1 Accepted 05 September 2005 Journal of Negative Results in BioMedicine 2005 4 7 doi 1477-5751 -4-7 This article is available from http content 4 l 7 2005 Godde et al licensee BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License http licenses by which permits unrestricted use distribution and reproduction in any medium provided the original work is properly cited. Abstract Apoptosis the programmed death of cells plays a distinct role in the etiopathogenesis of Multiple sclerosis MS a common disease of the central nervous system with complex genetic background. Yet it is not clear whether the impact of apoptosis is due to altered .