tailieunhanh - Báo cáo y học: "Human depression: a new approach in quantitative psychiatry"

Tuyển tập các báo cáo nghiên cứu về y học được đăng trên tạp chí y học General Psychiatry cung cấp cho các bạn kiến thức về ngành y đề tài: Human depression: a new approach in quantitative psychiatry. | Cocchi et al. Annals of General Psychiatry 2010 9 25 http content 9 1 25 REVIEW ANNALS OF GENERAL PSYCHIATRY Open Access Human depression a new approach in quantitative psychiatry Massimo Cocchi 1 2 3 LucioTonello1 3 and Mark M Rasenick4 Abstract The biomolecular approach to major depression disorder is explained by the different steps that involve cell membrane viscosity Gsa protein and tubulin. For the first time it is hypothesised that a biomolecular pathway exists moving from cell membrane viscosity through Gsa protein and Tubulin which can condition the conscious state and is measurable by electroencephalogram study of the brain s Y wave synchrony. Introduction The need for a deep radical turning point in the world of psychiatry is rapidly growing. Present diagnostic methods cannot continue to be considered acceptable because they are almost completely based on the psychiatrist s opinion which does not have an objective diagnostic technology and thus has a very high error rate. A debate is essential between the advocates of traditional diagnostic and therapeutic methods and advocates of emerging methods resulting from new discoveries. Major depressive disorder and other related and nonrelated psychiatric conditions are still characterised and defined by descriptive and non-biological criteria but it is hoped that we can adequately characterise this and other psychiatric disorders with the addition of new quantitative approaches. Human depression in the interpretation of an artificial neural network Following the theory that a biomolecular involvement of the cell could be an expression of a psychiatric disorder we have tried to understand and explain this phenomenon. The intention was to study the platelet fatty acids composition in normal and depressed subjects 1 because of their similarity to neurons 2-10 . Membrane platelet fatty acids of subjects with a clinical diagnosis of major depression versus apparently normal .