tailieunhanh - báo cáo hóa học: " Primary glia expressing the G93A-SOD1 mutation present a neuroinflammatory phenotype and provide a cellular system for studies of glial inflammation"

Tuyển tập báo cáo các nghiên cứu khoa học quốc tế ngành hóa học dành cho các bạn yêu hóa học tham khảo đề tài: Primary glia expressing the G93A-SOD1 mutation present a neuroinflammatory phenotype and provide a cellular system for studies of glial inflammation | Journal of Neuroinflammation BioMed Central Research Open Access Primary glia expressing the G93A-SOD1 mutation present a neuroinflammatory phenotype and provide a cellular system for studies of glial inflammation Kenneth Hensley 1 2 Haitham Abdel-Moaty1 3 Jerrod Hunter1 Molina Mhatre1 Shenyun Mou1 Kim Nguyen1 Tamara Potapova1 Quentin N Pye1 Min Qi1 Heather Rice1 Charles Stewart1 Katharine Stroukoff1 and Melinda West1 Address 1Free Radical Biology and Aging Research Program Oklahoma Medical Research Foundation OMRF 825 NE 13th Street Oklahoma City OK 73104 USA 2Department of Cell Biology University of Oklahoma Health Science Center OUHSC Oklahoma City OK 73104 USA and 3University of Oklahoma College of Engineering Bioengineering Program Norman OK USA Email Kenneth Hensley - kenneth-hensley@ Haitham Abdel-Moaty- haitham-abdel-moaty@ Jerrod Hunter - jerrod-hunter@ Molina Mhatre - molina-mhatre@ Shenyun Mou - shenyun-mou@ Kim Nguyen - kim-nguyen@ Tamara Potapova - tamara-potapova@ Quentin N Pye - quentin-pye@ Min Qi - min-qi@ Heather Rice - heather-rice@ Charles Stewart - charles-stewart@ Katharine Stroukoff- katharine-stroukoff@ Melinda West - melinda-west@ Corresponding author Published 25 January 2006 Received 01 September 2005 Journal ofNeuroinflammation 2006 3 2 doi 1742-2094-3-2 Accepted 25 January 2006 This article is available from http content 3 1 2 2006 Hensley et al licensee BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License http licenses by which permits unrestricted use distribution and reproduction in any medium provided the original work is properly cited. Abstract Detailed study of glial inflammation has been hindered by lack of cell culture .