tailieunhanh - Chapter 118. Infective Endocarditis (Part 3)

Clinical Manifestations The clinical syndrome of infective endocarditis is highly variable and spans a continuum between acute and subacute presentations. Native valve endocarditis (whether acquired in the community or in association with health care), prosthetic valve endocarditis, and endocarditis due to injection drug use share clinical and laboratory manifestations (Table 118-2). The causative microorganism is primarily responsible for the temporal course of endocarditis. β-Hemolytic streptococci, S. aureus, and pneumococci typically result in an acute course, although S. aureus occasionally causes subacute disease. Endocarditis caused by Staphylococcus lugdunensis (a coagulase-negative species) or by enterococci may present acutely. Subacute endocarditis is typically caused. | Chapter 118. Infective Endocarditis Part 3 Clinical Manifestations The clinical syndrome of infective endocarditis is highly variable and spans a continuum between acute and subacute presentations. Native valve endocarditis whether acquired in the community or in association with health care prosthetic valve endocarditis and endocarditis due to injection drug use share clinical and laboratory manifestations Table 118-2 . The causative microorganism is primarily responsible for the temporal course of endocarditis. 0-Hemolytic streptococci S. aureus and pneumococci typically result in an acute course although S. aureus occasionally causes subacute disease. Endocarditis caused by Staphylococcus lugdunensis a coagulase-negative species or by enterococci may present acutely. Subacute endocarditis is typically caused by viridans streptococci enterococci CoNS and the HACEK group. Endocarditis caused by Bartonella species and the agent of Q fever C. burnetii is exceptionally indolent. Table 118-2 Clinical and Laboratory Features of Infective Endocarditis Feature Frequency Fever 80-90 Chills and sweats 40-75 Anorexia weight loss malaise 25-50 Myalgias arthralgias 15-30 Back pain 7-15 Heart murmur 80-85 New worsened regurgitant murmur 10-40 Arterial emboli 20-50 Splenomegaly 15-50 Clubbing 10-20 Neurologic manifestations 20-40 Peripheral manifestations Osler s nodes subungual hemorrhages Janeway lesions Roth s spots 2-15 Petechiae 10-40 Laboratory manifestations Anemia .

• Y học thường thức
• Infective Endocarditis
• bệnh học và điều trị
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• tài liệu học ngành y
• Harrisons Internal Medicine
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• Aortic valve
• Erysipelothrix rhusiopathiae
• Cardiac arrhythmias
• Significant regurgitation
• Non bacterial thrombotic endocarditis
• Lung cancer
• Deep vein thrombosis
• Transthoracic echocardiogram
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