tailieunhanh - Chapter 118. Infective Endocarditis (Part 1)

Harrison's Internal Medicine Chapter 118. Infective Endocarditis Infective Endocarditis: Introduction The prototypic lesion of infective endocarditis, the vegetation (Fig. 118-1), is a mass of platelets, fibrin, microcolonies of microorganisms, and scant inflammatory cells. Infection most commonly involves heart valves (either native or prosthetic) but may also occur on the low-pressure side of the ventricular septum at the site of a defect, on the mural endocardium where it is damaged by aberrant jets of blood or foreign bodies, or on intracardiac devices themselves. The analogous process involving arteriovenous shunts, arterioarterial shunts (patent ductus arteriosus), or a coarctation of the aorta is. | Chapter 118. Infective Endocarditis Part 1 Harrison s Internal Medicine Chapter 118. Infective Endocarditis Infective Endocarditis Introduction The prototypic lesion of infective endocarditis the vegetation Fig. 118-1 is a mass of platelets fibrin microcolonies of microorganisms and scant inflammatory cells. Infection most commonly involves heart valves either native or prosthetic but may also occur on the low-pressure side of the ventricular septum at the site of a defect on the mural endocardium where it is damaged by aberrant jets of blood or foreign bodies or on intracardiac devices themselves. The analogous process involving arteriovenous shunts arterioarterial shunts patent ductus arteriosus or a coarctation of the aorta is called infective endarteritis. Figure 118-1 Vegetations arrows due to viridans streptococcal endocarditis involving the mitral valve. Endocarditis may be classified according to the temporal evolution of disease the site of infection the cause of infection or a predisposing risk factor such as injection drug use. While each classification criterion provides therapeutic and prognostic insight none is sufficient alone. Acute endocarditis is a hectically febrile illness that rapidly damages cardiac structures hematogenously seeds extracardiac sites and if untreated progresses to death within weeks. Subacute endocarditis follows an indolent course causes structural cardiac damage only slowly if at all rarely metastasizes and is gradually progressive unless complicated by a major embolic event or ruptured mycotic aneurysm. In developed countries the incidence of endocarditis ranges from to cases per 100 000 population per year and remained relatively stable from 1950 to 2000. While rates of congenital heart diseases remain constant other predisposing conditions in developed countries have shifted from chronic rheumatic heart disease to illicit IV drug use degenerative valve disease intracardiac devices and health care-associated .

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• Infective Endocarditis
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• Harrisons Internal Medicine
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• Cardiac arrhythmias
• Significant regurgitation
• Non bacterial thrombotic endocarditis
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• Transthoracic echocardiogram
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