tailieunhanh - ALZHEIMER'S DISEASE: ITS DIAGNOSIS AND PATHOGENESIS - PART 5

Các thay đổi Neuropathological trong basalis hạt nhân tương quan với các biện pháp lâm sàng của bệnh sa sút trí tuệ. Acta Neuropathol. 98, 186-196. Irina, A., Seppo, H., Arto, M., Paavo, Thánh, R., và Hilkka, S. (1999). tải fl-amyloid không bị ảnh hưởng bởi mức độ nghiêm trọng của bệnh tim mạch ở bệnh nhân | ALZHEIMER S DISEASE 207 Hu J. Miyatake F. Aizu Y Nakagawa H. Nakamura s. Tamaoka A. Takahash R. Urakami K. and Shoji M. 1999 . Angiotensin-converting enzyme genotype is associated with Alzheimer disease in the Japanese population. Neurosci. Lett. 27J 65-67. Hyman B. T. and Trojanowski J. Q. 1997 . Editorial on consensus recommendations for the postmortem diagnosis of Alzheimer disease from the National Institute on Aging and the Reagan Institute Working Group on Diagnostic Criteria for the Neuropathological Assessment of Alzheimer Disease. . Neumpathol. Exp. Neurol. 56 1095-1097. Iraizoz I. Guijarro J. L. Gonzalo L. M. and de Lacalle s. 1999 . Neuropathological changes in the nucleus basalis correlate with clinical measures of dementia. Acta Neuropathol. 98 186-196. Irina A. Seppo H. Arto M. Paavo Sr. R. and Hilkka s. 1999 . -Amyloid load is not influenced by the severity of cardiovascular disease in aged and demented patients. Stroke 30 613-618 Irizarry M. c. McNamara M. Fedorchak K. Hsiao K. and Hyman B. T. 1997a . APP-SW transgenic mice develop age-related A i deposits and neuropil abnormalities but no neuronal loss in CAI. . Neuropathol. Exp. Neurol. 56 965-973. Irizarry M. c. Soriano F. McNamara M. Fedorchak K. Hsiao K. and Hyman B. T. 1997b . ỉ ýí deposition is associated with neuropil changes but not with overt neuronal loss in the human amyloid precursor protein V717f PDAPP transgenic mouse. . Neurosci. 17 70537059. Iwata N. Tsubuki s. Takai Y Watanabe K. Sekiguchi M. Hosoki E. Kawashima-Morishima M. Lee H. J. Hama E. Sekine-Aizawa Y and Saido T. c. 2000 . Identification of the major A ỉ 1 42-degrading catabolic pathway in brain parenchyma Suppression leads to biochemical and pathological deposition. Nat. Med. 6 143-150. Iwatsubo T. Saido . Mann . Lee andTrojanowski . 1996 .Full-length amyloid- l l-42 43 and amino-terminally modified and truncated amyloid- 342 43 deposit in diffuse plaques. Am. J. Pathol. 149 1823-1830. Jack c. R. Petersen