tailieunhanh - Chapter 029. Disorders of the Eye (Part 10)

Central retinal artery occlusion combined with ischemic optic neuropathy in a 19-year-old woman with an elevated titer of anticardiolipin antibodies. Note the orange dot (rather than cherry red) corresponding to the fovea and the spared patch of retina just temporal to the optic disc. Figure 29-7 Hypertensive retinopathy with scattered flame (splinter) hemorrhages and cotton-wool spots (nerve fiber layer infarcts) in a patient with headache and a blood pressure of 234/120. Impending branch or central retinal vein occlusion can produce prolonged visual obscurations that resemble those described by patients with amaurosis fugax. The veins appear engorged and phlebitic, with numerous retinal hemorrhages. | Chapter 029. Disorders of the Eye Part 10 Central retinal artery occlusion combined with ischemic optic neuropathy in a 19-year-old woman with an elevated titer of anticardiolipin antibodies. Note the orange dot rather than cherry red corresponding to the fovea and the spared patch of retina just temporal to the optic disc. Figure 29-7 Hypertensive retinopathy with scattered flame splinter hemorrhages and cotton-wool spots nerve fiber layer infarcts in a patient with headache and a blood pressure of 234 120. Impending branch or central retinal vein occlusion can produce prolonged visual obscurations that resemble those described by patients with amaurosis fugax. The veins appear engorged and phlebitic with numerous retinal hemorrhages Fig. 29-8 . In some patients venous blood flow recovers spontaneously while others evolve a frank obstruction with extensive retinal bleeding blood and thunder appearance infarction and visual loss. Venous occlusion of the retina is often idiopathic but hypertension diabetes and glaucoma are prominent risk factors. Polycythemia thrombocythemia or other factors leading to an underlying hypercoagulable state should be corrected aspirin treatment may be beneficial. Figure 29-8 Ssynet F utl AS Ktcptr DL. 6r u v ld t h ui BL Lemgo DL Jjmttsn JL Lo c o J Hirrisvn t iw frqpiir af Zrtici 17th Ediboni http sim d iri . com Copyright o Th Nlcûriw-H II Comp n f Ens. All right ri rv d. Central retinal vein occlusion can produce massive retinal hemorrhage blood and thunder ischemia and vision loss Anterior Ischemic Optic Neuropathy AION This is caused by insufficient blood flow through the posterior ciliary arteries supplying the optic disc. It produces painless monocular visual loss that is usually sudden although some patients have progressive worsening. The optic disc appears swollen and surrounded by nerve fiber layer splinter hemorrhages Fig. 29-9 . AION is divided into two forms arteritic and nonarteritic. The nonarteritic form of .