tailieunhanh - MIG-6 suppresses endometrial epithelial cell proliferation by inhibiting phospho-AKT

Aberrant hyperactivation of epithelial proliferation, AKT signaling, and association with unopposed estrogen (E2) exposure is the most common endometrial cancer dysfunction. In the normal uterus, progesterone (P4) inhibits proliferation by coordinating stromal-epithelial cross-talk, which we previously showed is mediated by the function of Mitogen-inducible gene 6 (Mig-6). | MIG-6 suppresses endometrial epithelial cell proliferation by inhibiting phospho-AKT

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