tailieunhanh - Nocturnal penile erections: A retrospective study of the role of RigiScan in predicting the response to sildenafil in erectile dysfunction patients

Sildenafil enhances the nitric oxide–cGMP pathway of erection, which is claimed to have a role in nocturnal penile tumescence and rigidity (NPTR). This study aimed to find whether RigiScan can predict the response to sildenafil among erectile dysfunction (ED) patients and to find which RigiScan parameter produces the best prediction. Medical records of 172 ED patients were revised regarding their full sexual history, standard andrology examination, NPTR monitoring by the RigiScan device, and the degree of response to sildenafil. Of 172 ED patients, 94 patients () were sildenafil responders. All RigiScan parameters were higher in the sildenafil responder group. The RigiScan parameters with the most differentiating power between both sildenafil responders and non-responders were base rigidity (AUC ) and then tip rigidity (AUC ). The cut-off value of base and tip rigidity with the highest diagnostic accuracy was . This finding was found to be more specific than the sensitivity in predicting a positive response to sildenafil ( vs. and vs. , for base and tip rigidity, respectively). Sildenafil response in ED cases can be predicted through NPTR monitoring using the RigiScan device and ED patients with RigiScan base or tip rigidity less than 42% are not expected to respond well to sildenafil. | Nocturnal penile erections: A retrospective study of the role of RigiScan in predicting the response to sildenafil in erectile dysfunction patients

• Y khoa - Dược
• Erectile dysfunction
• Prognostic factors
• Nocturnal penile erections
• The role of RigiScan
• Predicting the response
• Erectile dysfunction patients
• scientific reports
• thesis report
• medical report
• Obesity is associated with a high prevalence of erectile dysfunction; how ever
• the pathophysiological link between obesity and erectile dysfunction remains poorly understood
• In this minireview
• we have attempted to eval uate the existing literature pertaining to obesity and erectile dysfunction to determine whether a common pathophysiological link exists
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