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Glycolysis is essential for chemoresistance induced by transient receptor potential channel C5 in colorectal cancer

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Elevated intracellular Ca2+ ([Ca2+]i ) level could lead to [Ca2+]i overload and promote apoptosis via different pathways. In our previously study, up-regulated expression of transient receptor potential canonical channel (TRPC5) was proven to increase [Ca2+]i level, and resulted in chemoresistance whereas not apoptosis in human colorectal cancer (CRC) cells. | Glycolysis is essential for chemoresistance induced by transient receptor potential channel C5 in colorectal cancer

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